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This review focuses on three key aspects: first, systematically dissecting the differential molecular pathways through which core OSA pathophysiological features (CIH, SF, sympathetic hyperactivity) regulate lung cancer, as well as their synergistic remodeling effects on key TME components (exosomes, tumor-associated macrophage polarization, cancer stem cells, and epigenetic modifications)
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[DOI] [PMC free article] [PubMed] [Google Scholar] Beelman RB, Kalaras MD, Richie JP
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